Skip to main navigation Skip to main content

CMH : Clinical and Molecular Hepatology

OPEN ACCESS
ABOUT
BROWSE ARTICLES
FOR CONTRIBUTORS

Articles

Reply to Correspondence

Reply to correspondence 1 on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”

Clinical and Molecular Hepatology 2026;32(1):e119-e120.
Published online: June 2, 2025

1Department of Applied Biology and Chemical Technology, The Hong Kong Polytechnic University, Hong Kong

2State Key Laboratory of Chemical Biology and Drug Discovery, The Hong Kong Polytechnic University, Hong Kong

Corresponding author : Terence Kin Wah Lee Department of Applied Biology and Chemical Technology, The Hong Kong Polytechnic University, Room 805, Block Y, Lee Shau Kee Building, Hong Kong Tel: +852-3400-8799, Fax: +852-2364-9932, E-mail: terence.kw.lee@polyu.edu.hk

Editor: Han Ah Lee, Chung-Ang University College of Medicine, Korea

• Received: May 13, 2025   • Accepted: May 30, 2025

Copyright © 2026 by The Korean Association for the Study of the Liver

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

  • 2,263 Views
  • 16 Download
next
Dear Editor,
I would like to express my appreciation to Tiantian Wang, Wenjie Huang and Limin Xia for their Correspondence [1,2] as a reply to my editorial entitled “Unveiling TRIB3: A New Mediator in MET-Driven Hepatocellular Carcinoma Progression” [3]. As a cancer researcher focused on basic liver cancer research, I found their insights particularly engaging. The exploration of TRIB3’s role in hepatocellular carcinoma (HCC) across different etiologies, including hepatitis B virus-related HCC and non-alcoholic steatohepatitis-related HCC, is indeed intriguing. Utilizing alpha-fold predictions to identify the molecular interactions between TRIB3 and COP1 may present a promising avenue for research. Based on this analysis, the authors may develop a therapeutic peptide [4] with the goal to disrupt the interaction between these proteins, thereby confirming their functional interplay. I believe the proposed future directions will provide a more comprehensive understanding of the role of TRIB3 in HCC. I would like to conclude this Reply by expressing my gratitude to the responses of Wang and the colleagues for providing further characterization, future perspectives and therapeutic implications for the role of TRIB3 in HCC.

Authors’ contribution

M.S.C drafted the manuscript. T.K.L reviewed and finalized the manuscript.

Acknowledgements

This work was supported by the Research Impact Fund (R5008-22F) and the RGC General Research Fund (15101621).

Conflicts of Interest

The authors have no conflicts to disclose.

HBV

hepatitis B virus

HCC

hepatocellular carcinoma

NASH

non-alcoholic steatohepatitis
  • 1. Wang T, Rao D, Fu C, Sun Z, Luo Y, Lu J, et al. MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation. Clin Mol Hepatol 2025;31:1032-1057.
  • 2. Wang T, Huang W, Xia L. Correspondence to Editorial 1 on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”. Clin Mol Hepatol 2026;32:e90-e92.
  • 3. Chan MSM, Lee TKW. Unveiling TRIB3: A new mediator in MET-driven hepatocellular carcinoma progression: Editorial on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”. Clin Mol Hepatol 2026;32:432-435.
  • 4. Li M, Wang Z, Tao J, Jiang H, Yang H, Guo D, et al. Fructose-1,6-bisphosphatase 1 dephosphorylates and inhibits TERT for tumor suppression. Nat Chem Biol 2024;20:1505-1513.

Download Citation

Download a citation file in RIS format that can be imported by all major citation management software, including EndNote, ProCite, RefWorks, and Reference Manager.

Format:

Include:

Reply to correspondence 1 on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”
Clin Mol Hepatol. 2026;32(1):e119-e120.   Published online June 2, 2025
Download Citation

Download a citation file in RIS format that can be imported by all major citation management software, including EndNote, ProCite, RefWorks, and Reference Manager.

Format:
Include:
Reply to correspondence 1 on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”
Clin Mol Hepatol. 2026;32(1):e119-e120.   Published online June 2, 2025
Close
Reply to correspondence 1 on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”
Reply to correspondence 1 on “MET promotes hepatocellular carcinoma development through the promotion of TRIB3-mediated FOXO1 degradation”