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Does limited expression of toll-like receptor 9 actually contribute to T cell activation and liver damage in nonalcoholic steatohepatitis? |
Hyuk Soo Eun |
Clin Mol Hepatol. 2020;26(2):236-237. Published online 2020 April 7 DOI: https://doi.org/10.3350/cmh.2020.0040 |
Does limited expression of toll-like receptor 9 actually contribute to T cell activation and liver damage in nonalcoholic steatohepatitis? Toll-like receptor 2 and palmitic acid cooperatively contribute to the development of nonalcoholic steatohepatitis through inflammasome activation in mice Limited expression of toll-like receptor 9 on T cells and its functional consequences in patients with nonalcoholic fatty liver disease Activation of the endotoxin/toll-like receptor 4 pathway: The way to go from nonalcoholic steatohepatitis up to hepatocellular carcinoma Hepatocyte mitochondrial DNA released in microparticles and toll‐like receptor 9 activation: A link between lipotoxicity and inflammation during nonalcoholic steatohepatitis Enhanced Expression of Toll-Like Receptor 4 and MyD88 is Associated With Disease Progression in Human Nonalcoholic Fatty Liver Disease Toll-Like Receptor 4 Activation by Damage-Associated Molecular Patterns (DAMPs) Mechanism regulating cell surface expression and activation of Toll-like receptor 4 Glucagon-Like Peptide-1 Receptor Agonists for Treatment of Nonalcoholic Fatty Liver Disease and Nonalcoholic Steatohepatitis: An Updated Meta-Analysis of Randomized Controlled Trials Small Intestinal Bacterial Overgrowth in Nonalcoholic Steatohepatitis: Association with Toll-Like Receptor 4 Expression and Plasma Levels of Interleukin 8 |
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